Six Quantum Biology Principles to Follow in 2026: As we enter a new year, it’s worth revisiting a set of biological fundamentals that govern human health. 1. See the sunrise consistently Morning sunlight sets the brain’s master clock (the suprachiasmatic nucleus) and synchronizes timing across every cell. This coherence supports metabolism, hormone signaling, mitochondrial energy production, and neural integrity. Without it, biology runs out of phase. 2. Rebalance indoor lighting Modern indoor environments are dominated by blue-heavy LEDs and lack the abundance of red and infrared wavelengths found in natural sunlight. Reintroducing broad-spectrum light using incandescent bulbs helps restore missing signals that support mitochondrial function and ATP production. 3. Get healthy, sequential UV exposure UVA and UVB play essential roles in neurotransmitter production, immune function, and vascular regulation. UV exposure also stimulates melanin production, which serves protective and detoxifying roles—binding heavy metals in the bloodstream and sequestering them in the skin for removal. Under UV light, melanin can dissociate water, generating a direct-current electrical flow and molecular hydrogen, a selective antioxidant that can cross the blood-brain barrier. What matters most is gradual, sequential exposure that trains the system rather than overwhelms it. 4. Block artificial blue light at night Blue light exposure after sunset suppresses melatonin and elevates cortisol at a time when the body should be shifting into repair mode. Protecting both the eyes and the skin—where melanopsin is also expressed—and maintaining darkness at night are foundational for cellular cleanup and regeneration through melatonin-regulated autophagy and apoptosis. 5. Reduce excess non-native EMF exposure Human exposure to non-native electromagnetic radiation has increased by a factor of one quintillion (that's a billion times a billion) in the last century. Reducing exposure—through distance, downtime, and duration—protects the mitochondria, cellular charge, and redox potential. 6. Eat seasonally and locally Food carries environmental signals that help the body determine whether to prioritize growth (mTOR) or repair (AMPK). Eating high-UV foods—especially sugars and carbohydrates—during low-UV seasons creates a mismatch with local light cues and increases deuterium burden, which can impair mitochondrial energy production. Aligning diet with season and latitude supports metabolic flexibility and mitochondrial efficiency. These simple, actionable inputs are what human biology requires to function optimally and activate the body’s innate healing intelligence. If you’re interested in deeper educational resources or individualized guidance, learn more at www.regenerint.com.

Modern Indoor Lighting Has Created a Public Health Crisis - Here’s Why Incandescent Bulbs Can Help 👇 Your indoor lighting environment has a tremendous impact on your health. Each wavelength (or color) of light plays a specific role in regulating your biology. Most people have no idea how dramatically modern lighting—and I mean just since the early 2000s—has altered the biological signals their bodies evolved to depend on. ➡️ Our Historical Light Environment (until very recently) Over the course of billions of years, life on Earth evolved under the full electromagnetic spectrum of natural sunlight — from invisible long-wavelength infrared, through the visible spectrum, to invisible short-wavelength ultraviolet. For most of human history, we effectively brought the outdoor light spectrum indoors. Fire, candlelight, and incandescent light bulbs are all thermal sources of light. When measured with a spectrometer, they emit a wide, balanced spectrum that is relatively low in short-wavelength light (such as blue) and smoothly increases into longer wavelengths (such as red), with abundant invisible infrared—which we experience as heat. This balance matters. Shorter-wavelength light carries more energy and is inherently stimulatory. In isolation, that stimulation can become stressful and damaging. In nature, however, short-wavelength light is always balanced by abundant long-wavelength light that is restorative. For reference, natural sunlight contains roughly (depending on location and time of year): ~8% ultraviolet (shorter wavelength) ~42% visible light (ROYGBIV) ~50% infrared (longer wavelength) If we were exposed only to long-wavelength light (red and infrared), we’d lack the stimulation needed to initiate steroid hormone production and mobilize energy for the day. If we were exposed only to short-wavelength light, energy production would suffer and aging would accelerate — our mitochondria would struggle to function. ➡️ The Evolutionary Shock of LED Lighting Everything changed abruptly in the early 2000s, when light-emitting diodes (LEDs) rapidly replaced incandescent bulbs. Unlike fire or the Sun, LEDs are not thermal light sources. They emit very narrow bands of visible light while removing infrared entirely. This was framed as an efficiency win — LEDs produce visible light without “wasting” energy as heat. But that so-called waste heat is biologically meaningful. Infrared light plays a critical role in: - Building exclusion-zone water inside and around our cells - Supporting mitochondrial energy production - Penetrating tissues to support repair processes Removing long-wavelength light while isolating high-energy short-wavelength light creates a signal that does not exist in nature — and one we are not adapted to. Chronic exposure to blue-enriched light without balancing red and infrared has been linked to: - Sleep and circadian disruption - Diabetes and metabolic dysfunction - Attention, focus, and cognitive issues - Myopia and macular degeneration - Systemic energy loss tied to mitochondrial stress ➡️ Infrared Light is an Essential Nutrient for Our Mitochondria Long-wavelength light penetrates deeply through tissues in a way short-wavelength light does not. Inside the body, infrared light interacts directly with mitochondria to support energy production. One mechanism involves water inside and around mitochondria. Long-wavelength light alters the viscosity of this water, allowing ATP synthase—the molecular turbine at the end of the electron transport chain—to spin more efficiently and generate more ATP. Additionally, cytochrome c oxidase (Complex IV of the electron transport chain inside the mitochondria) absorbs light in the near-infrared range, supporting metabolic water production. This metabolic water becomes charge-separated exclusion-zone water, which is critical for cellular energy production, cell membrane integrity, detoxification, and more. Infrared light also stimulates localized melatonin production inside mitochondria, where melatonin acts as a potent antioxidant—neutralizing excess reactive oxygen species that are a normal (and healthy) byproduct of oxidative phosphorylation. In short: long-wavelength light is an essential nutrient for mitochondrial function. And mitochondrial function determines the overall state of health. Modern indoor lighting has removed this nutrient almost entirely—while simultaneously increasing exposure to isolated short-wavelength light that promotes inflammatory and hypoxic conditions inside the body. ➡️ It’s Not Just LEDs — It’s Indoor Living Even without artificial lighting, modern windows are designed to block heat transfer — which means they filter out much of the red, infrared, and UV light while allowing proportionally more blue light to pass through. The result is an indoor environment that is blue-shifted by default. Combined with screens and LED lighting, we’ve created living spaces that bear little resemblance to the conditions under which human biology evolved. These alien light environments are affecting us, our children, our pets, our mental health, our physical health, and our future. ➡️ So What Can You Do? There is a widespread lack of physics education when it comes to light and its biological effects, and economic incentives often (usually) override public health considerations. As consumers, we need to drive the market and make our voices heard. Incandescent bulbs, while largely phased out, are still available for “decorative” use and remain the most biologically reliable indoor lighting option. As awareness grows, “circadian-friendly” or “full-spectrum” LEDs are entering the market. These can be useful tools, but it’s important to understand their limitations. LEDs still emit narrow wavelength spikes and generally lack infrared. They do not replicate the smooth, full spectrum emitted by natural thermal light sources. We’ve studied isolated wavelengths—but we know far less about how the full spectrum works together as an integrated biological signal. This is why the simplest, most affordable, and most reliable option remains the incandescent bulb (and a fireplace, if you have one). ➡️ Balancing Your Indoor Light Spectrum Incandescent bulbs can help you replicate a natural spectrum of outdoor light that your biology expects. Think of this as spectrum balancing. Indoors — between screens, LED light fixtures, and window-filtered daylight — you are exposed to a very blue-enriched environment lacking red and infrared. Your goal is to add those missing wavelengths back in. - During the day, incandescent bulbs can help counter overly blue indoor environments. Placing one near your workspace can provide supportive ambient long-wavelength light. - At night, red incandescent bulbs are preferable to red-only LEDs (because they contain infrared) for protecting circadian rhythms and preventing melatonin suppression. - Non-toxic beeswax candles and fireplaces are also excellent low-blue, long-wavelength light sources. - Circadian-friendly LEDs can be useful when incandescents aren’t an option — just remember they are still a compromise, not a replacement for thermal light. ➡️ A Seasonal Reminder As we move deeper into winter and the holiday season, people spend more time indoors under artificial lighting. That lighting can either be stress-inducing — subtly affecting mood, sleep, and behavior — or it can support calm, connection, and restoration. This season, consider your light environment and how it’s shaping the experience of everyone exposed to it. Use what we now understand. Reclaim your space. Support your biology. And bring a bit more of nature’s balance back into your home. ☀️

Life is a solar energy–dissipating structure. High-energy photons enter the system and are absorbed, emitted, reabsorbed, and re-emitted in a cascading process that ultimately releases lower-energy photons. This photonic cascade continuously excites electrons, activating molecules and atoms and enabling the downstream biochemical reactions that sustain life. Life on Earth is tuned to the electromagnetic spectrum of our Sun—from low-energy infrared to visible light to high-energy ultraviolet. When we deprive ourselves of the full spectrum, we deprive our biology of the inputs it evolved under, is adapted to, and depends upon for health, vitality, and coherent function. We are now facing a chronic disease epidemic of massive proportions. One of the greatest variables—and deepest root causes—is a widespread ultraviolet and infrared deficiency coupled with artificial blue-light toxicity, created by modern environments and lifestyles that disconnect us from the laws of nature—and from ourselves. It’s time to return to the light, reconnect with the source of life on Earth, and create environments that restore biological coherence rather than erode it.

Starving Lyme: How to Exploit Borrelia’s Metabolic Weakness A 2025 study published in mBio, a journal of the American Society for Microbiology, titled “Lactate dehydrogenase is the Achilles’ heel of Lyme disease bacterium Borreliella burgdorferi” revealed a key insight: the Lyme bacteria relies entirely on a single enzyme — lactate dehydrogenase (LDH) — for its metabolism and survival. The researchers concluded that this enzyme could be a promising drug target. But the same observations also point toward natural, non-pharmaceutical solutions. By understanding how Borrelia depends on lactate for its energy and redox balance, we can apply this knowledge to how we live, eat, and manage our metabolism to create unfavorable conditions for the bacteria. What the Study Found: The 2025 study highlighted that Borrelia burgdorferi has a highly restricted metabolism, relying solely on the anaerobic fermentation of glucose—glycolysis—for ATP production. To keep glycolysis running, the bacteria depend critically on the lactate dehydrogenase enzyme, which interconverts pyruvate and lactate, helping to recycle NAD⁺ and maintain the NAD⁺/NADH redox balance. Redox refers to reduction-oxidation, or the transfer of electrons between molecules. NAD⁺ acts as an electron acceptor, becoming NADH once it receives an electron. NADH then donates that electron to power metabolic reactions, converting back into NAD⁺ so the cycle can continue. Without lactate dehydrogenase, Borrelia loses the ability to regenerate NAD⁺, halting glycolysis — and therefore halting its only way of making energy. In essence, it can no longer recharge its redox batteries and quickly loses its charge. Borrelia’s Adaptability: Borrelia’s ability to run lactate dehydrogenase in both directions — converting pyruvate to lactate or lactate to pyruvate — is a key adaptation that enables it to survive in two completely different environments: the tick gut and the mammalian host. Inside the tick gut, glucose is scarce but lactate accumulates. Lyme bacteria import this lactate through a transporter called lactate permease (LctP), converting it into pyruvate for energy. Inside mammals, where glucose is abundant, the bacteria reverse this process — fermenting glucose into lactate to regenerate NAD⁺ and sustain glycolysis. This simple enzyme provides the metabolic adaptability that allows Borrelia to thrive in both environments. Human Comparison: Humans can also produce energy through glycolysis. But unlike Borrelia, we have mitochondria that provide another, far more efficient energy pathway. Inside the mitochondria, electrons from the breakdown of food are passed through the electron transport chain (ETC), generating an electrical current and proton gradient that drives the production of ATP and metabolic water — together creating the energy that powers life. This process, called oxidative phosphorylation, can run on electrons derived from carbohydrates, fats, or proteins, unlike glycolysis, which relies solely on glucose. When mitochondrial function breaks down — due to circadian disruption, diets high in processed and deuterium-loaded foods, chronic infections, or overexposure to mitochondrial stressors like artificial blue light and non-native electromagnetic fields (nnEMFs) — the cell reverts to glycolysis as a survival mechanism. This shift represents metabolic hypoxia, where the cell loses redox potential and oxygen efficiency. Glycolysis produces far less ATP, generates no metabolic water, and produces large amounts of lactate as a byproduct — creating the very conditions that Borrelia exploits. Targeting Lyme’s Vulnerability Through Diet: Knowing that Borrelia depends on glucose and lactate for its metabolism, and that it can parasitically import lactate through LctP, it stands to reason that environments low in both glucose and lactate are unfavorable for its survival. Our mitochondrial flexibility gives us the unique ability to create such an environment. When we shift into a ketogenic metabolic state, relying primarily on fats and ketones for energy, we activate mitochondrial respiration and move away from glycolysis. Two things happen simultaneously: There’s less glucose available for the bacteria to ferment. There’s less lactate produced for it to scavenge. A targeted, intelligently implemented ketogenic diet can therefore cut off Borrelia’s two main fuel lines — glucose and lactate. This doesn’t mean there’s zero glucose or lactate in the system, but their availability can be dramatically reduced, creating a metabolic terrain that stresses the bacteria and limits its growth. When chronic infection, inflammation, and hypoxia drive our own cells toward glycolysis, we essentially recreate the low-oxygen, high-lactate conditions of the tick gut inside our own bodies. Lyme disease thrives on the byproducts of our metabolic inefficiency. It exploits a terrain of low redox potential and poor mitochondrial function — the same terrain that underlies many chronic illnesses. By restoring mitochondrial health and returning to oxidative phosphorylation, we produce clean energy with no lactate output, starving Borrelia of the very resources it depends on. Restoring Mitochondrial Function: To shift out of glycolysis and back into mitochondrial energy production, the health of the mitochondria themselves — which have their own genome separate from the human nuclear genome — is foundational. Supporting mitochondrial health involves providing them with the environmental inputs they depend on while mitigating known mitochondrial stressors and toxins. This means aligning with natural circadian rhythms (morning sunlight exposure, blocking artificial blue light after sunset), reducing nnEMF exposure, grounding whenever possible, maintaining a low deuterium load, and ensuring regular exposure to infrared and full-spectrum sunlight — all of which help optimize the electron transport chain. Deuterium, for example, is a heavy isotope of hydrogen that is heavily concentrated in industrial seed oils, processed foods, and carbohydrates. It is twice as large and heavy as regular hydrogen (protium) and can jam the ATP synthase nanomotor at the end of the ETC, which is only designed to pump protium. This disrupts electron and proton flow, forcing metabolism back toward glycolysis. Infrared light powers Complex IV of the ETC, stimulating the production of deuterium-depleted metabolic water — which forms structured, charge-separated Exclusion Zone (EZ) water — and supporting local melatonin production, which is essential for managing oxidative stress and maintaining redox balance. The production of EZ water through oxidative phosphorylation is also critical, as layers of structured water inside and around our cells support detoxification and provide shielding against intracellular pathogens or membrane damage — including Borrelia’s scavenging of host membrane lipids, which it uses to cloak itself from immune recognition. In other words, fixing your circadian rhythm, getting plenty of natural full-spectrum and infrared-rich sunlight, avoiding excess artificial blue light and nnEMF (which can create hypoxic conditions), and strategically implementing ketosis all help deplete the body of excess deuterium and restore mitochondrial electron transport efficiency. This reactivates oxidative phosphorylation — enabling you to produce energy cleanly, rather than through glycolysis, which generates the lactate that Lyme uses as fuel. The Seasonal Diet Connection: A best practice to support metabolic health is to consume a diet that is both local and seasonal to where you live. At northern latitudes, as sunlight and UV exposure drop in winter, carbohydrate availability naturally declines. Ancestrally, this would have shifted our metabolism into a mild, fat-based ketogenic state — an AMPK-dominant phase focused on repair, autophagy, and regeneration. In summer, with higher UV exposure and carbohydrate abundance, metabolism shifts toward mTOR activation, promoting growth, protein synthesis, and energy storage. Both phases are essential. Today, artificial light environments and the constant availability of imported, out-of-season foods keep us stuck in perpetual summer — high glucose, high lactate, high deuterium, and chronic inflammation. We cut ourselves off from the restorative processes of winter — autophagy, ketosis, and deuterium depletion. Returning to seasonal patterns — higher fat intake in winter, higher carbohydrate intake in summer — helps restore the metabolic rhythms that support mitochondrial health and keep pathogens like Borrelia in check. The Takeaway: The 2025 study by Sze et al. identified Borrelia’s dependence on lactate dehydrogenase as its metabolic weakness — its Achilles’ heel. While the researchers proposed that lactate dehydrogenase could serve as a target for developing pharmaceutical drugs that impair Borrelia’s metabolism, the same observation reveals another, more immediate path: restore mitochondrial function and shift metabolism away from glycolysis. We have mitochondria and can run on fats and ketones where Lyme cannot. This is where our advantage lies. By focusing on mitochondrial health and producing energy through oxidative phosphorylation, we can create a coherent metabolic terrain in which Lyme cannot thrive. CITE: Sze CW, Lynch MJ, Zhang K, Neau DB, Ealick SE, Crane BR, Li C. Lactate dehydrogenase is the Achilles' heel of Lyme disease bacterium Borreliella burgdorferi. mBio. 2025 Apr 9;16(4):e0372824. doi: 10.1128/mbio.03728-24. Epub 2025 Mar 20. PMID: 40111021; PMCID: PMC11980376. Looking for Support in Healing from Lyme? If you’re navigating Lyme or biotoxin illness, the path forward isn’t always made easier by adding another prescription — it begins by rebuilding coherence. I’ve been through it, and know it isn’t easy. My Lyme & Biotoxin Illness Recovery Package is designed to restore redox potential, repair mitochondrial function, and help you clear environmental and microbial burdens by focusing on building a strong biophysical foundation. Learn more at www.regenerint.com

Biotoxins from mold, Lyme, and other sources can lead to chronic inflammation, immune dysfunction, and hormonal imbalances - particularly in individuals with HLA-DR genetic susceptibility. Roughly 25% of the population carry specific HLA-DR gene variants that impair the immune system’s ability to clear these toxins. Once bound to bile, they get reabsorbed and recirculate, keeping the body in a chronic inflammatory loop. Over time, this drives excess cytokine production, which blocks leptin signaling at proopiomelanocortin (POMC) neurons in the hypothalamus. When POMC signaling is disrupted, production of alpha-melanocyte stimulating hormone (α-MSH) drops—reducing the body’s ability to regulate inflammation, immune balance, metabolism, and gut integrity. Low α-MSH and impaired POMC signaling are linked to: • Chronic inflammation and fatigue • Immune dysregulation (MARCoNS, Candida, etc.) • Gut permeability and food sensitivities • Adrenal dysfunction and fibromyalgia-like pain Recovery requires more than removing exposure—it means restoring cellular communication between light, leptin, and POMC signaling, addressing inflammation at its biophysical roots.

What is Redox Potential?
Understanding the Body’s Healing Capacity What is Redox Potential? “Redox potential” refers to the body’s ability to maintain a balance of electrons available for transfer in reduction-oxidation (redox) reactions. These reactions involve the exchange of electrons between two species (atoms or molecules): one species gains electrons (reduction), while the other loses electrons (oxidation). A high redox potential means the body has a sufficient reserve of electrons to support essential processes like energy production, detoxification, and cellular repair. Why is Redox Potential Important for our Biology? Redox reactions (the transfer of electrons) drive cellular energy production, facilitate communication between cells, support detoxification, and help manage inflammation and the repair of tissues. When the body has a sufficient store of electrons (a high redox potential), it can effectively manage these processes and maintain overall health. However, if redox potential is low due to a lack of available electrons, the body struggles to manage oxidative stress, leading to cellular dysfunction, impaired healing, and a higher risk of inflammation and disease. Redox and Inflammation Inflammation is like a controlled fire the body uses to clear out infections or damaged cells. This process occurs in two phases. Phase One uses oxidants to steal electrons and “set the fire”, breaking down microbial pathogens, toxins, or irreparable cells. Once this is complete, Phase Two begins, where antioxidants donate electrons to neutralize the oxidants and "put out the fire." When both phases are completed, debris is cleared and balance is restored, allowing for new, healthy growth. However, if redox potential is low, the body may struggle with Phase Two, leading to a slow, uncontrolled burn and chronic, low-level inflammation with ongoing symptoms. Redox and Energy Creation Cellular energy production in the mitochondria relies on a continuous cycle of redox reactions involving two key electron carriers, NADH and FADH₂, which act like buckets transporting electrons derived from the breakdown of food. NADH and FADH₂ donate these electrons to the electron transport chain, where they are funneled through a series of protein complexes to drive the production of cellular energy. By giving up electrons, NADH and FADH₂ become oxidized, turning into NAD+ and FAD—the "empty buckets" ready to accept new electrons (becoming reduced) and continue the cycle. Redox and Detoxification In the liver, enzymes use oxidation (stealing electrons) to convert fat-soluble toxins into water-soluble forms that can be excreted from the body. After this, glutathione, a powerful antioxidant, uses reduction (donating electrons) to neutralize these toxins, ensuring their safe elimination. Redox and Cellular Communication Reactive oxygen species (ROS)—molecules with unpaired electrons—are key signaling agents that help cells communicate about internal and external conditions. This signaling is essential for coordinating processes like immune responses, cell growth, and adaptation to stress. When redox potential is high, ROS signaling is balanced, enabling proper cellular communication and function. Increasing Redox Potential with Exclusion Zone Water Exclusion Zone (EZ) water is a structured, negatively charged, gel-like form of water inside the body that forms inside and around our cells and tissues. EZ water’s unique structure creates a reservoir of electrons that the body can use for redox reactions. Because EZ water surrounds all our fascia and connective tissue, electrons can be instantaneously transported anywhere throughout the body where they may be needed. The more EZ water we have, the more electrons we can store, enhancing our redox potential and overall healing capacity. How to Increase Redox Potential: Gather Electrons Food: Metabolizing food releases electrons stored in fats, proteins, and carbs to be used in the body. Grounding/Earthing: The surface of the Earth is an infinite repository of electrons, which flow into our bodies with direct skin contact. Sunlight Exposure: Infrared light supports the expansion of EZ water inside the body, and also stimulates the mitochondria to produce more metabolic water which then becomes EZ water. Exposure to ultraviolet light also generates free electrons stored in the EZ. Physical Activity: Exercise generates electrons through piezoelectric effects, where mechanical pressure creates an electric charge. How to Increase Redox Potential: Minimize Electron Loss Address Indoor Lifestyle: Our species evolved to be continuously connected to the Earth's natural sources of electrons and full-spectrum sunlight. Disconnection from nature leaves us reliant primarily on food for electrons, leading to widespread electron deficiency. Mitigate Exposure to Non-Native Electromagnetic Fields (nnEMFs): WiFi, cell phones, and wireless devices have been shown to collapse EZ water by about 15%, lowering redox potential. Wireless radiation also causes calcium to flood into cells, disrupting mitochondrial function and their production of metabolic water. Limit Environmental Toxins: Exposure to pesticides, herbicides, heavy metals, and other toxins increases the body’s demand for electrons to neutralize these substances, reducing redox potential.

The Biotoxin Illness Pathway & Implications of Reduced α-MSH: What Are Biotoxins? Biotoxins are toxic substances of biological origin, released by sources like mold, Lyme disease, and other tick-borne illnesses. In most people, the immune system can effectively identify, break down, and eliminate biotoxins from the body. This means that once they are removed from the source — such as a water-damaged building with mold mycotoxins — the body can naturally detoxify itself. Biotoxin Susceptibility Approximately 25% of the population has a genetic susceptibility to biotoxin illness due to specific HLA-DR genetic variants. These variants impair the immune system's ability to recognize and eliminate biotoxins, even after the source of exposure is removed. This explains why some individuals become chronically ill from biotoxin exposure while others remain unaffected. Biotoxin Accumulation For individuals with genetic susceptibility, biotoxins can be difficult to eliminate and may accumulate in the body. In the liver, biotoxins bind to bile, which is sent to the gastrointestinal tract for excretion. However, during the process of enterohepatic circulation, bile is reabsorbed in the small intestine and returned to the liver. In individuals with the HLA-DR genetic variant, the biotoxins are not effectively removed and are reabsorbed along with the bile. This recycling process leads to persistent symptoms, even after the original source of exposure has been eliminated. Cytokine Production & Impaired Leptin Signaling Biotoxins get stored in fat cells, triggering the release of excess inflammatory cytokines. These cytokines can block leptin receptors on proopiomelanocortin (POMC) neurons in the hypothalamus, preventing leptin from binding effectively. Without proper leptin signaling, POMC activity is disrupted, inhibiting its production of critical peptides like alpha melanocyte-stimulating hormone (α-MSH), which helps regulate inflammation, immune function, metabolism, and more. Implications of Reduced α-MSH Chronic Inflammation: α-MSH reduces inflammation by suppressing pro-inflammatory cytokines. Without sufficient α-MSH, excess cytokines can lead to fatigue, headaches, muscle pain, and difficulty concentrating. Immune Dysfunction: α-MSH plays a key role in immune balance. Its deficiency leaves the body vulnerable to things like candida overgrowth and colonization of mucus membranes by antibiotic resistant bacteria (MARCoNS), which releases endotoxins that further inhibit α-MSH. Gastrointestinal Disorders: α-MSH protects the integrity of the gut lining. Low α-MSH can contribute to symptoms of “leaky gut”, like bloating, food sensitivities, and IBS-like symptoms. Adrenal Fatigue & Fibromyalgia Inflammatory cytokines triggered by biotoxin illness initially cause an increase in ACTH and cortisol production. However, over time, chronic inflammation and disrupted hypothalamic signaling leads to a significant reduction in these hormones, contributing to symptoms of adrenal fatigue. This is further exacerbated by disrupted circadian rhythms. Impaired leptin signaling at POMC neurons in the brain also reduces the production of beta-endorphin, an endogenous opioid. This can cause increased sensitivity to pain and the symptoms associated with fibromyalgia. Clearing Biotoxins 1. Addressing the Source: It’s important to remove the source of biotoxin exposure, whether through mold remediation or treating tick-borne infections. 2. Binding and Eliminating Toxins: Biotoxins are negatively charged, which means positively charged binders may be the most effective at drawing them out of the body. 3. The Importance of Natural Light: UV light stimulates POMC cleavage, producing α-MSH and beta-endorphin. Safe UV exposure requires the development of a healthy solar callus, starting with morning infrared light to prime the skin. 4. Minimizing Artificial Blue Light & nnEMF: Excessive artificial blue light exposure can shift POMC cleavage toward ACTH production, while certain nnEMF wavelengths may inhibit the ability of α-MSH to produce melanin. 5. Supporting Circadian Rhythms: Morning sunlight exposure and blocking artificial light at night helps regulate the body’s circadian rhythms, which are critical for immune function, leptin signaling, melatonin production, and much more. 6. Supporting Detoxification: Supporting the expansion of coherent domains/Exclusion Zone water inside the body (via proper hydration, grounding, and infrared light) pushes toxins out of cells and into the interstitial fluid, where they can be bound and eliminated. If you're struggling with biotoxin illness, work closely with your healthcare professional to evaluate your situation and determine the next steps. Recovering from mold, Lyme, and other biotoxin-related illnesses can be a complex journey. At REGENERINT, I offer ongoing support and consultations from a biophysics-based perspective. Reach out at REGENERINT.com to learn how you can support your path to recovery with sustainable lifestyle and environmental changes.

Why the Food You Eat Should Match Your Light Environment ☀️ The food you eat is encoded with information about the light environment in which it grew. Our bodies rely on light as a primary cue to regulate our daily circadian rhythms and our seasonal infradian rhythms. When the light information you consume through food doesn’t align with the light information that your eyes and skin detect, it confuses your body’s ability to determine the time of year and the appropriate metabolic program to run. This mismatch can lead to metabolic stress and chronic inflammation. By eating foods that are seasonally and locally available, you ensure that the environmental signals your body receives are coherent, supporting a healthier and more balanced metabolism. Photosynthesis & Electron Excitation: All food webs begin with the capture and storage of light. During photosynthesis, plants transform photonic energy into chemical energy, effectively alchemizing light into the physical matter that fuels their growth. This transformation occurs due to the excitation of electrons by photons. When photons of light strike chlorophyll molecules in plant leaves, the electrons of these molecules absorb the photon’s energy and jump to higher orbital levels around the nucleus of the atom they orbit. This excitation creates potential energy which is transferred to a reaction center inside the chloroplast, where it powers the conversion of water and carbon dioxide into glucose and oxygen. Food as an Electromagnetic Barcode: Different photons carry varying levels of electromagnetic energy. Higher-energy photons, such as ultraviolet (UV) light, excite electrons to higher orbital states. In this way, the specific wavelengths of sunlight absorbed by a plant are imprinted onto the electrons within the plant matter. As a result, food acts like an electromagnetic barcode—a unique marker of the light environment in which it grew. Just as a barcode contains information about a product, the electrons in plant matter carry information about the light conditions that produced them. When we consume these foods, we’re not just ingesting calories or nutrients; we’re also taking in this stored light information. Biochemical Responses to Light: Foods contain different chemical compounds depending on the light energy levels they are exposed to during growth. High UV exposure triggers plants to produce photoprotective compounds like certain polyphenols, carotenoids, and anthocyanins. While the electrons in chlorophyll molecules will eventually return to their lower energy states after absorbing high-energy UV photons, the UV-adaptive compounds the plants produced remain. These compounds reflect the specific light conditions under which the plants grew, and act as another form of stored light information. Light Modulates the Gut Microbiome: Light exposure, particularly UVB light, has been shown to influence the gut microbiome. Seasonal changes in light—such as longer days with higher UV exposure in summer versus shorter days with less UV in winter—affect which bacteria thrive in our gut and the types of digestive enzymes our body produces. In summer, increased UV exposure and abundant carbohydrates promote a microbiome optimized for digesting fruits and plant-based sugars. In winter, with lower UV exposure and nutrient scarcity, the gut microbiome shifts to better support the digestion of fats and proteins. How Light Informs Metabolic Programs: When we consume food, we ingest information about the light environment in which the food grew. As our mitochondria metabolize food, the light information is released, informing our cells about external conditions. This enables our bodies to regulate metabolic programs according to seasonal cycles and the availability of light. Summertime conditions — marked by longer daylight hours, abundant nutrients, and increased ATP production — activate the mTOR pathway, an anabolic process that promotes growth and the buildup of energy stores. Wintertime conditions — marked by shorter daylight hours, higher melatonin levels, nutrient scarcity, and reduced ATP production — activate the AMPK pathway, a catabolic process that breaks down energy stores and promotes autophagy. While mTOR fosters a growth-focused, inflammatory state, AMPK supports an anti-inflammatory state that prioritizes energy conservation and cellular repair. Issues with Eating Non-Local, Out-of-Season Foods: Consuming out-of-season imported foods creates a mismatch between the light signals your body receives from the environment and the light information in your food. If your body is experiencing winter (low UV light) but you eat tropical foods grown in high UV conditions, you send mixed signals about which metabolic program to run, making it harder to prioritize growth or repair. Your gut may also lack the bacteria and enzymes to digest these foods due to light’s influence on the microbiome. Over time, chronically overactivating the mTOR pathway by mimicking perpetual summer can lead to inflammation, insulin resistance, weight gain, and accelerated aging. Benefits of Eating Seasonally and Locally: Eating foods that contain light information that matches your current environment provides your body with coherent signals to help determine which metabolic programs to run—whether to store or burn fat, grow or repair tissues. Seasonal eating also supports digestion, as shifts in the gut microbiome are influenced by changing light conditions. By embracing nature's seasonal cycles rather than resisting them, you allow your body to fully switch between metabolic states, promoting metabolic flexibility and enhancing mitochondrial health. This gives your body the opportunity to complete its natural cycles, entering a state of winter regeneration instead of remaining in a constant state of summertime inflammation. #quantumbiology #seasonaleating #metabolichealth