Are you not entertained?

Many people have functional deficiencies that are being overlooked by only focusing on absolute deficiencies in their labs. Great thing about metabolomics testing is that the imbalances often tell you about functional deficiencies. Intake is one thing. Absorption is another. Then there is assimilation, utilisation, depletion and potential blockages to enzymatic activity (& genetics) as well.

𝗟𝗶𝗴𝗵𝘁 𝗶𝘀 𝗻𝗼𝘁 𝘀𝘁𝗮𝘁𝗶𝗰. 𝗜𝘁 𝗰𝗵𝗮𝗻𝗴𝗲𝘀 𝘁𝗵𝗿𝗼𝘂𝗴𝗵𝗼𝘂𝘁 𝘁𝗵𝗲 𝗱𝗮𝘆 𝗶𝗻 𝗮 𝘄𝗮𝘆 𝘁𝗵𝗮𝘁 𝗽𝗿𝗼𝗳𝗼𝘂𝗻𝗱𝗹𝘆 𝗶𝗻𝗳𝗹𝘂𝗲𝗻𝗰𝗲𝘀 𝗵𝘂𝗺𝗮𝗻 𝗯𝗶𝗼𝗹𝗼𝗴𝘆. Understanding these changes and how they compare to artificial light sources is key to optimizing your health. Using spectrometry, we can observe these shifts and see why early morning natural light exposure is crucial for synchronizing our internal clocks. At sunrise, the intensity of blue light rapidly increases, reaching the same level as red and near-infrared light. UVA first enters the spectrum of light we receive when the sun is roughly 10 degrees above the horizon. And the UVB rise takes place at 30 degrees. During solar noon peak, the blue portion of the spectrum becomes dominant. At this time, blue light intensity surpasses other wavelengths, acting as a powerful daytime signal to the body. This reinforces wakefulness, cognitive alertness, and metabolic function. This is why you want to get outside as often as possible throughout the day, to give your mitochondrial colony & physiological systems the accurate inputs it requires to function optimally.

I like exercising outside. Between sets of pullups/dips/pushups/squats etc, I walk around for a minute or two. If my workout is 20 sets, then that is 40 minutes of walking (5,000 steps) integrated into my workout. Alternatively, Ill do some mobility work between sets. Grounded and in the sun. This is the way.

I truly think conventional medicine does not understand Type 2 Diabetes, and that is why its solutions are so unsuccessful. Type 2 diabetes is not primarily a problem of high blood sugar but of impaired mitochondrial energy utilization. Glucose and fatty acids are both broken down into acetyl-CoA, which feeds into the TCA cycle. The cycle generates electron carriers, NADH and FADH2, which deliver electrons and protons to the ETC. When functioning well, electrons flow down the ETC to oxygen, protons are pumped across the inner mitochondrial membrane, and ATP is produced efficiently through oxidative phosphorylation. This coupling between substrate oxidation and energy demand keeps the system in balance. This is basic mitochondrial biology we all learn in med school (but is never integrated into working with people). In metabolic disease, however, the mitochondria are flooded with fuel. Chronic excess of glucose, free fatty acids, and triglycerides means the cell receives far more input than it can oxidize. But the issue is also often combined with impaired oxidation itself for which the bottleneck can be found in many different places. This is what doesn't get enough attention. Both fats and sugars converge on the same mitochondrial machinery, and when the influx of electrons from NADH and FADH2 exceeds the ETC’s capacity to pass them to oxygen, the chain becomes over-reduced (flooded with electrons essentially). You get hyperpolarization of the mitochondrial membrane. Electrons begin to leak from complexes I and III, generating oxidative stress. When produced excessively high levels of ROS will damage mitochondrial proteins, lipids, and membranes, impairing oxidative capacity further - vicious cycle as with most chronic diseases. The oxidative stress will blunt insulin signalling itself. Meanwhile, build up of diacylglycerol and ceramides due to fuel overload reinforce insulin resistance. This creates a self-perpetuating loop of mitochondrial stress and signalling dysfunction. The cell, in essence, becomes insulin resistant as a defence mechanism. When mitochondria are already overloaded, the cell reduces glucose uptake to prevent further electron pressure and oxidative damage. Systemically, this appears as hyperglycaemia and hyperinsulinemia, but the problem began within the mitochondria, not the bloodstream. It is an issue of energy oversupply and disposal, not merely sugar toxicity. At its core, type 2 diabetes is a mitochondrial problem, a breakdown in the handling of electrons, not simply a surplus of glucose. The blood abnormalities are the visible signs of this deeper bioenergetic congestion - but they are not the disease itself. So you should not optimise these markers as you can “fix” those while making the issue worse.

They don't talk about glyphosate in medical training. Eating organic is seen as a hippie thing with no purpose. They are wrong.

Anatomy as Optics The body can be seen as a living optical system designed to distribute light intelligently. - The skin acts as a filter and diffuser. - The CSF serves as a light guide for the brain. - The blood vessels act as photon absorbers and transporters of NO-mediated signals. - The eye/sclera facilitate infrared light to the retina - The amniotic fluid ensures that the fetus is immersed in light. The optical properties of our tissues are not accidental. They are tuned by evolution to channel the wavelengths that sustain metabolism, regulate oxidative balance, and synchronize cellular rhythms with the external world. Nature has built optics into our biology. All you have to do is live a life in accordance with it.